The scientific community acknowledges the heart health benefits of soy, a claim validated in 1999 by the Food and Drug Administration. Scientists knew soy-based diets reduced LDL (bad) cholesterol and triglycerides while raising HDL (good) cholesterol.

Studies show that lunasin, a naturally occurring peptide is largely responsible for soy’s cardiovascular benefits.  LunaRich soy powder and the further concentrated LunaRich X™ optimize bioactive lunasin content for maximum benefit to you.

Cholesterol Reducing Dual-Action LunaRich

Cholesterol comes from two sources: dietary intake and our body’s own production in the liver. Over 80% of  comes from the liver, so the most effective way to lower LDL (bad) cholesterol is to control the liver’s internal production. The lunasin in LunaRich does so in two ways:

  1. Selectively disrupts a step in the production of an enzyme key to cholesterol synthesis in the liver.
  2. Increases the number of receptors available in liver cells to clear LDL cholesterol from the bloodstream.

Most cholesterol is produced in the liver by the HMG-CoA reductase gene. Simply blocking HMG-CoA can lead to health problems because your body needs a minimum level of cholesterol for proper cellular function. Lunasin instead targets cholesterol at an earlier stage, reducing the HMG-CoA without blocking it entirely. So you get cardiovascular benefits without disrupting the body’s normal functioning.

Natural Reduction of Cholesterol with LunaRich Flyer (PDF)

Learn more about the health benefits of lunasin


AHA Recognizes Lunasin’s Heart Health Benefits

Lunasin, the key ingredient in Reliv’s LunaRich® whole bean non-GMO soy powder, and its ability to support cardiovascular health have been recognized by the American Heart Association! Circulation the official journal of the AHA, has published a study abstract demonstrating lunasin’s heart health benefits.

Lunasin’s Heart Health Studies

Meta-analysis of the effects of soy protein containing isoflavones on the lipid profile.

RGD-peptide lunasin inhibits Akt-mediated NF-κB activation in human macrophages through interaction with the αVβ3 integrin.

Endocytic mechanism of internalization of dietary peptide lunasin into macrophages in inflammatory condition associated with cardiovascular disease.